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Comprehensive toxicology evaluations reveal that the compound does not affect food intake, body temperature regulation, or basic physiological parameters at effective doses in animal models, indicating that its metabolic effects occur through specific pathway modulation rather than general physiological disruption

In contrast, sodium bicarbonate increases blood alkalinity by raising bicarbonate concentration, which improves buffering capacity against exercise-induced acidosis

Manipulating central nervous mechanisms of food intake and body weight regulation by intranasal administration of neuropeptides in man

If the abovementioned is occurring, it could mean that IGF-1 administration could also lower glucose levels by suppressing hepatic gluconeogenesis, as well as improving insulin signalling in this organ, leading to IGFBP-1 suppression and overall improvement of the IGF-1/GH/insulin axis

Results The results (see Figs