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Finally, inhibition of PGC-1 abolished the protective effects of AMSCs against APAP-induced mitochondrial damage

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The results showed that the GSH concentrations of deletion strain were higher than that of parental strain, and this phenotype could be restored in the complementation strain C gsh-px (Figure 7), suggesting that L

Emerging evidence supports that the CD38 ectoenzyme and products controlled by the CD38/NAD + axis may favor the onset of lung immunopathology

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