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The trouble is that a plausible pathway diagram is not evidence

[DOI] [PMC free article] [PubMed] [Google Scholar] 104.Garg A., Lee J.C

National Toxicology Program, Research Triangle Park, NC

2017 Jul;103:89-100

Indeed, the rise of endogenous GSH leads to elevated SA levels ( PR-1a in foliar tissues ( PR-1 expression was also observed in a lesions simulating disease ( lsd ) mutant of Arabidopsis thaliana that spontaneously develops HR-like necrotic lesions ( PR-1a expression to a similar extent as SA, in both Xanthi NN and SA-deficient NahG leaves, although gene induction is slower and less pronounced in NahG plants ( Figure S3 )

The activity is mediated by MT3, a quinine-reductases