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CD56 (NCAM-1): HLA-independent targeting in neuroendocrine malignancies Another CAR-T approach, developed by Crossland DL

Cancer cells inherently produce excess ROS under rapid metabolism, making them more sensitive to GSH depletion

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Reduced concentrations of 2-oxoglutarate and acetoacetate along with an upregulation of creatinine, pyruvic acid, glycerophosphocholine and serine emerged as biochemical blood signatures in PD patients

CSCs are a subpopulation of tumor cells with the capability to self-renew and differentiate, which drives tumor progression and metastasis

An additional finding in one of the studies was that maintenance doses were required to sustain the effect of the TSO treatment (Summers et al ., )