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Apart from external inducers, endogenous triggers such as mast cell-derived histamine, proteases and eicosanoids can increase intestinal permeability, either directly or via stimulation of neurons of the enteric nervous system [194],[199],[200]

These anomalies occur because the kidneys and the brain share parts of their structure, have similar systems for controlling blood flow, and communicate through chemicals and other signals (Bugnicourt et al., 2013