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Reductive Stress Creates Oxidative Stress An experiment 5 used heart-specific overexpression (TG) or deletion (DN) of NOX4 (NADPH Oxidase 4) in mice to examine its effects on Ischemia (heart attack) injury versus normal mice (WT)

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Interestingly, the effects of AOA could be overridden by treatment with dimethyl-alpha-ketoglurate (DMKG), suggesting KG or its downstream metabolite mediates the effects of glutamate

This scenario could result from decrements in mitochondrial bioenergetics, including a decreased supply of GTP for autophagic vesicular processing

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In support of these two major take a-ways, (pH dependent dissolution and reversion to more stable and lower solubility polymorphs) are the studies that explored the apparent solid forms