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Description

Mitochondrial dna depletion, oxidative stress, and mutation: mechanisms of dysfunction from nucleoside reverse transcriptase inhibitors

Gip, L., Lodin, A

(5956:) T-lymphocyte activation antigen CD80 precursor (Activation B7-1 antigen) (CTLA-4 counter-receptor B7.1) (B7) (BB1)

1F) and DNA fragmentation assays (Fig

Howlin, P

In glioblastoma, autophagy facilitates chemoresistance by altering cellular metabolism, inducing quiescence, and enhancing survival, with transcriptome profiling uncovering both known oncogenic pathways and new genes possibly associated with glioblatoma development and therapeutic resistance (53)