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The investigators interpreted the effects as direct pharmacological validation of the NNMT-knockdown phenotype reported earlier in genetic models

[1] [3] These injections are prescription-only medicines (POMs) typically prescribed by GPs for patients with confirmed B12 deficiency

The worsening of symptoms, or the emergence of new symptoms following exertion, provides a valuable opportunity to investigate the underlying metabolic dysfunction associated with these conditions

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These results suggested that RGSH could ameliorate AKI by inhibiting ferroptosis, which indicates RGSH as a promising therapeutic strategy for the treatment of AKI

If conservative and regenerative options fail, surgical repair may be consideredespecially in cases of full-thickness tears or functional decline