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The results (Figure 5) showed that FVII depletion caused an increase in the thrombin generation kinetic parameters (i.e

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Lactate-induced mitochondrial ROS production activates the p38-SGK1 pathway, weakening neural precursor cell expressed, developmentally down-regulated 4-like E3 ubiquitin protein ligase (NEDD4L)s interaction with GPX4, contributing to etoposide resistance [59]