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Semax (BDNF/TrkB pathway, neuroplasticity) and the Selank (GABAergic pathway, anxiolysis) act through non-competitive and complementary mechanisms

This activity may be driven by increased levels of brain-derived neurotrophic factor (BDNF) or through a more direct modulation of specific receptors that govern cognitive processes

Whether this is optimal or simply reflects empirical practice isnt established

[Google Scholar] 105.Boschert V, Klenk N, Abt A, Janaki Raman S, Fischer M, Brands RC, Seher A, Linz C, Mller-Richter U, Bischler T, Hartmann S

Experimental systems investigating Epithalon often evaluate oxidative stress markers alongside telomere dynamics