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MITOCHONDRIAL MECHANISMS OF DbCM Both our increased understanding of mitochondrial mechanisms contributing to DbCM and the increasing elucidation of mechanisms of action of antidiabetic drugs has increased the attention for mitochondria as a therapeutic target in diabetic heart disease

doi: 10.1508/cytologia.75.443 30 FuloriaSMehtaJChandelASekarMRaniNNIMBegumMYet al

Acetylation limits 53BP1 association with damaged chromatin to promote homologous recombination

These trials should prioritize determining the optimal dosages, administration protocols and the effects of combination therapies

See Figure 1 for the variables