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Conclusion In summary, the present review outlined the dual role of ferroptosis in LC, highlighting both its detrimental and therapeutic effects, and biological agents that target ferroptosis as potential therapies for LC, with certain compounds inducing ferroptosis and decreasing tumor cell resistance

Platinum-induced peripheral neurotoxicity: from pathogenesis to treatment

Taken together, these results show that LOsG downregulates FoxO1, which releases its suppression effects on TXNIP

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