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Animal models are inherently costly, and the underlying mechanisms do not always translate to human disease processes ( in vitro 2D and 3D models are vastly different from the in vivo environment, making it difficult to fully understand the various factors affecting the neurovascular unit and pathways and mechanisms contributing to neuroinflammation

Another apparently general host response to bacterial pathogens is the activation of the transcription factor HIF-1 by stabilization of the HIF-1 subunit through hypoxic conditions caused by infections or pathogen-induced signaling (Rupp et al., 2007

The effect of integrating genomic information into genetic evaluations of chinese merino sheep

Amino Acids, 2012, 2012, 1-26

Blood samples All animals tested were randomly chosen from controlled farms of the Latium and Tuscany countrysides

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