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Yang et al, 1992

It stimulates lipolysis (fat breakdown) and inhibits lipogenesis (new fat cell formation) via beta-3 adrenergic receptor pathways, without elevating IGF-1 or disrupting glucose metabolism, an important distinction as perimenopausal women are already at increased risk for insulin resistance

Cells overexpressing AKT-E17K or AKT-WT differed neither in the basal NADPH/NADH ratio (untreated cells) nor in NADPH/NADH ratio determined 12 h after irradiation