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To confirm that YAP regulates GS expression in mouse brain astrocytes, we crossed YAP flox/flox mice 25 and Aldh1l1-CreER T2 mice 26 to generate Aldh1l1-CreER T2 :YAP flox/flox mice, and selectively knocked out YAP in astrocytes upon tamoxifen administration (Fig

To this end, we first investigated whether NHR-49 activity could recover upon refeeding

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Both complete loss-of-function and reduced function variants may result in a reduction of treatment efficacy, with atypical metabolite formation (e.g., switching from coumarin 7-hydroxylation to 3-hydroxylation) (Hadidi et al., 1997