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Rescue of cells from apoptosis by inhibition of active GSH extrusion

This framework thereby bridges molecular mechanisms to clinical disease, demonstrating oxidative stress as a unifying axis in multifactorial pathologies

Surprisingly, mice with specific genetic deletion of Gpx1 appear phenotypically normal and with normal life span (Ho et al., 1997), suggesting that there are alternative compensatory mechanisms for H 2 O 2 scavenging in Gpx1 deficiency

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[DOI] [PMC free article] [PubMed] [Google Scholar] 109.Montero AJ, Diaz-Montero CM, Deutsch YE, et al

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