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Severe hepatocellular reaction resembling alcoholic hepatitis with cirrhosis after massive small bowel resection and prolonged total parenteral nutrition

A CRISPR-based screen identifies genes essential for West-Nile-virus-induced cell death

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A recent study reported that activation of AMPK1 promoted PASMCs proliferation [23]

Whereas, aldehyde-reactive alkoxyamines capture AP sites, and inhibit the activity of APE1, the enzyme responsible for base excision repair (BER)

assessing a panel of six stimuli showed that monocyte-derived macrophages from individuals with CD were more resistant to apoptosis induced by phorbol 12-myristate 13-acetate (PMA) or TNF compared to healthy controls: they suggested that the prolonged presence of these macrophages would contribute to inflammation and limit tissue repair [9]