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Bump EA, Al-Sarraf R, Pierce SM, Coleman CN

In animal studies, it appears to influence pathways linked to VEGF, a key signal involved in healing [10], [11]

In vivo assessment of riluzole as a potential therapeutic drug for spinocerebellar ataxia type 3

357 Similarly, in HD and related polyQ disorders, expanded polyQ-containing proteins form insoluble aggregates that physically inhibit proteasome function, amplifying the proteotoxic burden

doi: 10.1097/MPG.0000000000002708 289

InczefiOBacquiVOlier-PierreMRincelMRingot-DestrezBEllero-SimatosSet alTargeted intestinal tight junction hyperpermeability alters the microbiome, behavior, and visceromotor responses