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As discussed previously, a tentative example of selective effect would be modulation of S1PR2 in astrocytes, which seems to be linked to glutamate recycling and mitochondrial metabolism (116), both promising mechanisms to ameliorate excitotoxic synaptopathy

These regions were chosen because of their putative role in the neural mechanisms of ME/CFS and long COVID, in particular with regard to the integration of information from various levels [33, 34], with pgACC key for emotional regulation and autonomic integration, and dACC - for cognition [35, 36]