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Taken together, our results suggest a conserved influence of intermediate filament association with aggresomes and neuronal extrusions that eject potentially toxic material

Following cell treatment with proteasome inhibitors, stably expressed ATG12 binds to and inactivates the antiapoptotic proteins Bcl-2 and Mcl-1 via a BH3-like motif, resulting in BAX activation, mitochondrial release of cytochrome c, and enhanced chemosensitivity [84]

TMAO is implicated in inflammation and oxidative stress, cholesterol metabolism, bile acid metabolism, and foam cell formation in AS [47]

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Hoppel, Quantification of carnitine and acylcarnitines in biological matrices by HPLC electrospray ionization-mass spectrometry, Clinical Chemistry 54 (9) (2008) 1451-1462

(2020), Liu et al