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In parallel, causal in vivo studies targeting key structural regulators of these contact sites, such as MFN2 and other tethering proteins, will be particularly informative for determining whether disruption of organelle interaction hubs directly contributes to mitochondrial dysfunction, metabolic imbalance, and functional ovarian decline

In agreement with these observations, an increase in the reduced form of plasma homocysteine associated with an elevation of the oxidation rate of homocysteine and cysteine (expressed as a decreased ratio of plasma reduced to total aminothiols) was reported in experimental hyperhomocysteinemia (Guttormsen et al, 1993

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J Strength Cond Res 37(7):e413e421 Wang Y et al (2023) Astaxanthin promotes mitochondrial biogenesis and antioxidant capacity in chronic high-intensity interval training

10.1161/ATVBAHA.120.315322 83