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Mitochondrial metabolic heterogeneity in CRC: implications for therapeutic divergence and precision intervention Building on earlier discussions of how mitochondrial metabolism contributes to resistance in CRC, driven by metabolic enzymes, intratumoural heterogeneity, and post-transcriptional regulatory networks, this section highlights the clinical relevance of metabolic divergence

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Although acting as miRNA sponges represents a well-established function of lncRNAs (144-146), their other regulatory functions are also implicated in cataract pathogenesis

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