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Intravenous injection of urocortin 1 induces a CRF 2 mediated increase in circulating ghrelin and glucose levels through distinct mechanisms in rats

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In these cases, observational designs, such as prospective cohort studies that track naturally occurring risk factors over time, may provide stronger or more ethical evidence

These models can be used not only to verify the pathogenicity of PD-related disease causative genes, but also to find appropriate interventions and therapeutic approaches

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Notably, plasma lipoproteins, as key mediators of systemic metabolism and inflammatory states, may play a pivotal bridging role in this process [18]