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doi: 10.1038/s41586-023-06568-6 19 JurgensMClausMWingertSNiemannJAPicardLKHennesEet al

97 Moreover, ceruloplasmin is a physiologic inhibitor of MPO, 98 a neutrophil enzyme that promotes oxidative stress in COPD

FIGURE 10 C75 and baicalin can directly activate CPT1A (Tomita et al., 2005), and the ACC2 inhibitors TOFA and Soraphen ( However, overexpression of CPT1A may increase the FAO rate, promote cell metabolism, and accelerate cell apoptosis, which brings certain difficulties for the development of related drugs