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While MenSC-Exos exert anti-inflammatory and antiapoptotic effects (Dalirfardouei et al., 2019), HBOT reduces neuronal apoptosis by downregulating the expression of proinflammatory cytokines (e.g., IL-1 and TNF-) (Ghaemi et al., 2023

Cu(I)-glutathione complex: a potential source of superoxide radicals generation

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Gut microbiota-derived metabolites, like SCFAs, were shown to play a pivotal role in the MGBA by elevating pro-inflammatory cytokines in the subjects' blood, worsening radiation-induced brain injury (Luo et al., 2022

Experimental models demonstrate that upregulating EAAC1 function enhances cysteine uptake, elevates GSH levels, and significantly reduces neuronal apoptosis and infarct size following ischemic insult