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According to Shah et al., enzymatic lipid peroxidation reactions, rather than non-enzymatic lipid peroxidation, play a crucial role in achieving the critical threshold of phospholipid hydroperoxides (PLOOH) for ferroptosis (Shah et al., 2018

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Moreover, as reported in a previous study 11,12 , ferroptotic cell death is capable of releasing its intracellular components as pro-inflammatory damage-associated molecular patterns (DAMPs), and the releasing and accumulation of DAMPs can trigger tissue inflammation, resulting in the development of various inflammatory diseases (e.g., PE 12 )

parkeri at a MOI of 1, and BSO was replaced at 1 hpi