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Several currently used cancer drugs exert their anticancer activity in part through covalent inhibition of TrxR 21 and two covalent TrxR inhibitors are currently in clinical trials 68

On the other hand, it induces cell cycle arrest and activates apoptotic pathways, thereby inhibiting tumor cell proliferation and promoting programmed cell death [233, 234]

The 42 amino acids that make up A42 are primarily degraded amyloid precursor protein, which can be aberrantly deposited outside of neuronal cells

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IB is then degraded, releasing the NF-B dimer from the cytoplasm into the nucleus, where it binds to DNA and promotes its transcription