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NAC cysteine compounds (in the reverse adjustment) play an important role in the synthesis of glutamate, with a portion provided by NAC cysteine by the sodium-dependent transport mechanism through the bloodbrain barrier, converted into cystine in the brain, and the latter provided via the cystine-glutamate transporter exchange to glutamate, which causes mGLuR2/3 receptor activation and leads to decreased synaptic glutamate release

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Most illnesses affected by inflammation become a target for the use of Tb4 in peptide therapy