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CuNPs can trigger mitochondria-mediated apoptotic pathways in the kidneys against oxidative stress [150]

Consistent with the latter, the suppressive effect of GSH on metHb formation should not be disregarded (57)

The advanced lipid peroxidation products generated from acrolein induced mitochondrial dysfunction can further deplete antioxidants such as SOD, GSH, and glutathione peroxidase impairing the antioxidant defense

Can these injuries heal on their own

The study identified a regulatory axis (VDAC3/DHODH) that enhances GSH-mediated ferroptosis resistance, demonstrating significant cardioprotective outcomes in vivo (Wang J