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Here, we present a novel mechanism by which TNBC cells of the MSL/CL subtype activate an OS response through direct induction of the NRF2 pathway by Cyss uptake, and we propose NRF2 and its direct transcriptional target OSGIN1 as interesting targets for personalized cancer treatment

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J Infect (2012) 65:58891

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Together, these two mechanisms help to minimize the loss of retinal ganglion cells, thereby slowing the progression of glaucoma (141)