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T cells not only contribute to maintaining liver tolerance, but are also important participants in liver damage and inflammation [153,154,155]

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In a sepsis-induced AKI model, USP9x inhibits the ubiquitinated degradation of toll-like receptor 4 (TLR4), which is involved in inflammatory responses and apoptosis by activating the NF-B signaling pathway through interaction with lipopolysaccharide (LPS)