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Apolipoprotein E4 (APOE 4), a major genetic risk factor for AD, disrupts endosome-lysosome and autophagy pathways, leading to impaired autophagy initiation, lysosomal dysfunction, and subsequent A and tau accumulation

Flautner and A

Neuregulin 4 suppresses NASH-HCC development by restraining tumor-prone liver microenvironment

Interplay between activation of endogenous retroviruses and inflammation as common pathogenic mechanism in neurological and psychiatric disorders

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