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Drug-induced liver injury (DILI) is a leading cause of acute liver failure and can be life-threatening

Molecular mechanism studies revealed that EAS upregulates PGC-1 expression, promotes mitochondrial biogenesis, and reduces ROS generation

Prolonged receptor activation through long-acting design: Native amylin is rapidly cleared from circulation, limiting its biological effect

Melideo, SL

[12] [13] Readers should note that final peer-reviewed results from this programme are still emerging, and specific percentage figures may change as the full dataset is published

Additionally, GLP-1 analogs reduced energy expenditure, with only tirzepatide showing a significant decrease in the respiratory quotient (RQ) in MC4R KO mice