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While these rapid responses occur legitimately, they should not create unrealistic expectations

Reduced sirtuin activity in an NAD + -depleted environment contributes, both indirectly via metabolic collapse and augmented fibrotic signaling, as well as by further inducing cellular senescence, to unchecked fibroblast activation and myofibroblast persistence driving intractable fibrosis

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Correlations between gene modules and experimental conditions (genotype or treatment) were tested using the limma R package, with multiple testing corrections via the BenjaminiHochberg method