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monocytogenes controlled its biphasic life cycle and transitioned from saprophytic to pathogenic by regulating the concentrations of GSH (Reniere et al., 2015)

115 This finding reinforces the role of DNA methylation profiling in optimizing treatment regimens

When sulforaphane enters the cell, it interacts with Keap1, causing it to release Nrf2

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Briefly, dried samples were resuspended in 100 l of 50 mM ammonium acetate (AA), after which excess NEM was removed using 300 l of dichloromethane

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