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The Hepatotoxicity Interaction: Additive Risk on a Stressed Liver Isotretinoin and high-dose GTE each carry independent DILI potential

Then, THOC3 was ablated or overexpressed in intestinal organoids (Fig

Acknowledgements This study is supported, in part, by the University of Kansas Medical Center Endowment Fund, and by research grants from Shenzhen Key Laboratory of Steroid Drug Discovery and Development (grant # ZDSYS20190902093417963) and the National Natural Science Foundation of China (grant # 81630096)

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Conclusion PUM2 overexpression suppressed oxidative stress and promoted autophagy to alleviate renal injury in DM-CIAKI by interacting with HDAC9 mRNA, which mediated HDAC9 and mRNA degradation and inhibited HDAC9 expression

Bilasy et al., 2015)