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British Journal of Clinical Pharmacology, 65 (1), 51-59

How BPC-157 Modulates Gastric and Esophageal Healing Pathways BPC-157's primary mechanism involves upregulation of VEGF and fibroblast growth factor (FGF), both of which drive angiogenesis

The activation is linked to the generation of vasoconstrictive agents, including MCP-1, (pro)renin receptor, endothelin, and angiotensin II, while concomitantly diminishing vasodilatory compounds like nitric oxide, which may contribute to the development of hypertension and lead to decreased viability of cardiomyocytes and myocardial damage