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This finding has significant implications for wound healing research in radiation-compromised tissue models

10.3389/fnins.2018.00245 Front

Further mechanistic studies showed that NNMTi increases intracellular NAD levels and activates the SIRT1 pathway by inhibiting NNMT, thereby suppressing the transcription of adipogenic genes and preventing the maturation of preadipocytes.This research provides a new perspective for understanding the molecular mechanism of adipocyte differentiation, confirms the key role of NNMTi in regulating adipogenesis, and lays a foundation for subsequent obesity intervention studies

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406 Several studies have shown that follicular B (FO B) cells can serve as progenitors for ABCs, and their findings further revealed that neither MHC-II-deficient nor CD40-deficient FO B cells are capable of generating ABCs