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Low-grade systemic inflammation driven by several mediators previously debated, such as LPS, bacterial translocation and the increased proinflammatory cytokines and Th1 response, are all contributing to an immunological response in the brain [139]

Quercetin treatment effectively mitigated these apoptotic changes in a dose-dependent manner, normalizing the BAX/ BCL-2 ratio, reducing CYTOCHROME C release, and suppressing CASPASE-3 activation

mice deficient in NLRP3 exhibit increased susceptibility to systemic candidiasis (Skeldon and Saleh, 2011)

Scriver CR, Beaudet AL, Sly WS, Valle D (eds) (New York, NY: McGraw-Hill) Franken DG, Boers GHJ, Blom HJ, Trijibels FJM and Kloppenborg PW (1994) Treatment of mild hyperhomocysteinemia in vascular disease patients

[DOI] [PMC free article] [PubMed] [Google Scholar] 74.Cesarone M.R., Belcaro G., Hu S., Dugall M., Hosoi M., Ledda A., Feragalli B., Maione C., Cotellese R