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While many medications may offer cardiovascular benefits by deregulating the RAAS, they can cause concurrent hyperkalemia because the RAAS facilitates potassium excretion in the kidneys

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Among B cells, B1 and marginal zone subsets, characterized by high CD36 expression are more susceptible to ferroptosis and rely on GPX4 for survival and antibody production, whereas follicular B cells are relatively ferroptosis-resistant due to lower intracellular fatty acid levels [136]

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