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6 Conclusion and outlook The intricate ferroptosis-immune-metabolic axis has emerged as a central driver of COPD pathogenesis, creating a self-perpetuating cycle of inflammation, oxidative stress, and tissue damage

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Understanding the roles and interventions of the GSH system, as well as its connection with ferroptosis, may favor the advancement of more effective therapies to prevent myocardial injury (summarized in Fig

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The increased expression of UPR target genes, including ATF4 , DDIT3 , TRIB3 , GADD34 , uXBP1 , and sXBP1 , identified by RNA-seq, was validated in MNNG/HOS and U2OS cell lines using RT-qPCR (Fig