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Our study found that chronic hypoxia promoted HIF-2 rather than HIF-1 entry into the nucleus, and nucleation of HIF-2 resulted in transcriptional expression of Acer2, a newly discovered downstream target of HIF that was reported in adipocytes [19, 24]

Andrew Huberman explained the mechanism on Rogan's podcast: BPC-157 acts like a "repair signal" that encourages fibroblasts, the cells that build connective tissue, to migrate to injury sites and start rebuilding

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