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Genome-scale metabolic modeling reveals that Telaglenastat is uniquely suited to counter the adaptive metabolic reprogramming induced by Palbociclib To obtain a systems-wide perspective of the metabolic reprogramming underlying treatment with Palbociclib, Telaglenastat, or their combination, we integrated the transcriptomics and metabolomics data from the residual xenograft tumors in the framework of Recon3D

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