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The disturbance of endoplasmic reticulum Ca2+ triggered by oxidative stress may also induce UPR and apoptosis ( + T cells to produce adaptive immune response, and T cells release interferon- (INF-), It binds to receptors on keratinocytes, further releases and presents inflammatory cytokines such as CXC-L16 and IL-15, and further recruits T cells to the skin through a positive feedback loop ( + and T cells induced by cytokines and chemokines ensure the final destruction of epidermal melanocytes (Figure 1)

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This must tax regulatory systems that prevent abnormal redox cycling of iron, such as the Fe 2+ -glutathione complexes thought to be the dominant form of iron in the cellular labile iron pool (Berndt and Lillig, 2017

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