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Consequently, VRK2 expression emerges as a dual-function biomarker: its targeted inhibition shows therapeutic efficacy in VRK2-high malignancies, while its deficiency serves as a predictive indicator for metabolic-targeted interventions

This dysregulation can lead to increased erythroid apoptosis and further exacerbate the iron overload condition, creating a vicious cycle of damage (78)

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These structural differences suggest that selective inhibition of TGR over human TrxR may be attainable

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Nitric oxide release in human aortic endothelial cells mediated by delivery of amphiphilic polysiloxane nanoparticles to caveolae