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Similar content being viewed by others Main The activation of nucleic-acid-sensing endosomal Toll-like receptors (TLRs) and late endosomal-initiated signaling are central mechanisms in innate immunity, inflammation and autoimmunity 1,2,3,4,5,6,7

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Rather than directly scavenging free radicals, GHK-Cu appears to upregulate genes encoding protective enzymes like superoxide dismutase [PMID: 29986520, 26236730]

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