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The pathogenesis of POI is complex and multifactorial and may be caused by defects in primordial follicle formation, follicle recruitment/distribution, follicle growth and development [46, 47]

Zhang W, Wang T, Xue, et al

10.3233/JAD-140396 446 TarangeloA.RodencalJ.KimJ

J Neuroinflammation 17(1):147

The reaction consumes the universal cellular methyl donor SAM and the NAD + salvage precursor nicotinamide, producing a metabolite (1-MNA) that is excreted in urine