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Conclusion The findings of this study support the potential of XSJ as a therapeutic agent for MASH, revealing its synergistic mechanisms involving multiple components, targets, and signaling pathways

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Microfold (M) cells: important immunosurveillance posts in the intestinal epithelium

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It was described that possible responsible pathomechanisms leading to CV complication development are triggered by ACE2, which is a key enzymatic component of the renin-angiotensin-aldosterone system (RAAS)