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The ability of OligoG to chelate calcium has been hypothesized as a mechanism by which it is able to modify the assembly of cystic fibrosis intestinal mucus and disrupt the extracellular polymeric network of mucoid P

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Glucagon-like peptide-1, but not glucose-dependent insulinotropic peptide, inhibits glucagon secretion via somatostatin (receptor subtype 2) in the perfused rat pancreas